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In severe cases of epidermolysis bullosa, local therapy is supplemented with systemic corticosteroids.

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  • The information is generalized and is provided for informational purposes only. Seek medical attention at the first sign of illness. Self-medication is dangerous to health!
  • Hereditary diseases that manifest vesicular eruptions without inflammatory symptoms are combined and are currently considered as different variants of one nosological form - epidermolysis bullosa (epidermolysis bullosa hereditaria).
  • The disease is detected in the first days of a child's life and only sometimes can be detected at a later age.
  • The development of methods for clinical, cytological and biochemical diagnosis of genodermatoses made it possible to distinguish two variants of epidermolysis bullosa - simple and dystrophic.
  • Etiology and pathogenesis. The causes leading to the inheritance of epidermolysis bullosa have not yet been established.
  • It is believed that this dermatosis occurs due to genetic coding of mutational changes in germ cells. It is noticed that in different families any one type of epidermolysis bullosa is inherited.

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A simple form of epidermolysis is characterized by the appearance of blisters at the sites of mechanical injuries, bruises, friction, pressure, even if they are minor. Boys are predominantly ill. On apparently healthyAuger without concomitant and previous inflammatory changes, blisters of various sizes are formed with serous, serous-purulent or hemorrhagic contents.

Bubbles usually occur on protruding areas: on the hands, front surfaces of the upper and lower extremities, on the feet, legs, buttocks. In infants, blisters develop around the mouth. The mucous membranes of the oral cavity and genital organs are rarely affected, only in 2-3% of patients. Changes in teeth, hair and nails are not observed or they appear very rarely.

Bubbles can be the size of a pea, walnut, or larger. They open quickly, exposing juicy, rapidly epithelizing erosions without scarring. Subjectively, there is itching or burning at the sites of blisters and erosions. The general condition of patients does not change. The process tends to buy levaquin spontaneously by the age of puberty.

Simple and dystrophic-hyperplastic forms of dermatosis are inherited in an autosomal dominant manner, and dystrophic-polydysplastic and lethal forms are inherited in an autosomal recessive manner.

  • A special form of simple epidermolysis bullosa is the summer variant of Weber-Kokkain.
  • The disease is characterized by the appearance of blisters only on the hands and feet in spring and summer, in combination with hyperhidrosis and keratosis of the palms and soles.
  • The hyperplastic form of dystrophic epidermolysis bullosa is formed in utero and is detected in the first days of a child's life.

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In places subject to trauma, blisters appear, and then erosion and ulcers with fragments of levaquin online. The lesion ends with dystrophic scars, sometimes of a keloid nature. The protruding areas of the skin are most often affected - the anterior surfaces of the elbow and knee joints, where, against the background of atrophic areas of lilac-red color, blisters with hemorrhagic contents and scaly-cortical layers appear that are tightly adjacent to the skin. When scales and crusts are removed, areas of cicatricial atrophy are exposed.

The skin of unaffected areas is dry, grayish-pale in color with persistent white dermographism. On the palms and soles are hyperhidrosis, hyperkeratosis. The nails of the feet and hands are thickened and deformed. Nikolsky's symptom is positive. In 20% of patients, the mucous membranes of generic levofloxacin, genital organs, and conjunctiva are affected. In these places, the bubbles quickly open. The remaining erosion epithelialize sluggishly, often turning into superficial ulcers. After healing, cicatricial atrophy and foci of leukokeratosis remain. In areas of atrophy, epidermal cysts such as whiteheads may occur.

The hyperplastic form is often combined with dental dystrophy, hypertrichosis or ichthyosis. The course of the disease is benign with an exacerbation in the summer. The process can be exacerbated in winter under the influence of thermal influences (hot bath, warm clothes, shoes).

A variant of hyperplastic dystrophic epidermolysis bullosa that appears along with blisters of miliary whitish papules is called white papuloid epidermolysis. There are small lichenoid-type miliary papules of a whitish-grayish hue, merging into large diffuse foci with micropolycyclic outlines. In places, along with the blisters, milia-like cysts are also formed. Rashes occur mainly on the feet and hands in the lumbosacral regions and on the extensor surface of the elbow and knee joints. The mucous membranes may be the site of small miliary papules and milia-like cysts resembling leukoplakia or leukokeratosis. Onychogryphosis and hypertrichosis are also observed.

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The polydysplastic form of epidermolysis bullosa is less common than the simple form (1 in 300,000 newborns). Numerous large blisters with serous-hemorrhagic contents occur at birth or in the first days of a child's life. Rashes are located on any part of the skin, even on the scalp, not affected by other varieties. Arising spontaneously, even without previous trauma, they are still more often on the limbs. Blisters are located deeper under the epidermis and, when opened, often form ulcerative surfaces complicated by pyogenic infection. Healing always occurs with deforming scars.

As a result of extensive cicatricial changes, ectropion of the eyelids, deformities of the mouth, hands and feet with contractures and mutations occur. Nikolsky's symptom is positive. In many cases of the disease, the mucous membrane of the mouth, as well as the pharynx, esophagus, trachea and genital organs are involved in the process.